1033 Central Neurogenic Hyperventilation May Cause Metabolic Alkalosis in Mitochondrial Encephalopathy
نویسندگان
چکیده
منابع مشابه
Lymphomatoid granulomatosis presenting as central neurogenic hyperventilation.
A patient with lymphomatoid granulomatosis and focal transformation to lymphoma limited to the central nervous system presented with severe central neurogenic hyperventilation. The hyperventilation resolved as the underlying pathologic condition was treated with prednisone and cyclophosphamide.
متن کاملRecurrent Primary CNS Lymphoma Presenting with Central Neurogenic Hyperventilation
Background: Tumor-induced Central Neurogenic Hyperventilation (CNH) is exceedingly rare, about half resulting from Primary CNS Lymphoma (PCNSL) despite their rarity. Results: We present a 75 year-old, immunocompetent woman with recurrent PCNSL leading to CNH. She was initially diagnosed with PCNSL via biopsy of the single enhancing left caudate focus among diffuse, nonenhancing CNS infiltrative...
متن کاملB-cell lymphoma of the brainstem with central neurogenic hyperventilation.
Non-Hodgkin's lymphoma of the brainstem is a rare entity. Central Neurogenic Hyperventilation (CNH), an associated manifestation of this disease, is an even rarer event. We report a case of an immunocompetent individual who presented to us with tachypnea and facial nerve palsy. Neuroimaging showed a Cerebellopontine angle tumour which on histopathology showed feature consistent with a Non-Hodgk...
متن کاملCentral neurogenic hyperventilation: a case report and discussion of pathophysiology.
BACKGROUND Central neurogenic hyperventilation is a rare condition with poorly understood pathophysiology. OBJECTIVE To describe a patient with central neurogenic hyperventilation caused by an infiltrative brainstem lymphoma. CONCLUSION Based on analysis of this patient and other case reports, we propose that central neurogenic hyperventilation is uniquely the result of infiltrative tumors ...
متن کاملA rare cause of hypokalaemia and metabolic alkalosis
A 72-year-old man with a medical history of diabetes mellitus Type II, paroxysmal atrial fibrillation and heart failure was hospitalized after a syncope. Pitting oedema in both legs were the only abnormal finding on physical examination, in particular the patient had no cushinoid features. The blood pressure was slightly elevated at 160/90 mmHg. Regular medications consisted of furosemid 100 mg...
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ژورنال
عنوان ژورنال: Archives of Disease in Childhood
سال: 2012
ISSN: 0003-9888,1468-2044
DOI: 10.1136/archdischild-2012-302724.1033